13,464 research outputs found

    Dispersion corrections to parity violating electron scattering

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    We consider the dispersion correction to elastic parity violating electron-proton scattering due to \gammaZ exchange. In a recent publication, this correction was reported to be substantially larger than the previous estimates. In this paper, we study the dispersion correction in greater detail. We confirm the size of the disperion correction to be 6% for the QWEAK experiment designed to measure the proton weak charge. We enumerate parameters that have to be constrained to better than relative 30% in order to keep the theoretical uncertainty for QWEAK under control.Comment: 6 pages, 3 figures, 2 tables; To be published in the proceedings of the VIII Latin American Symposium on Nuclear Physics and Applications, December 15-19, 2009, Santiago, Chiil

    Quenching and Tomography from RHIC to LHC

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    We compare fully perturbative and fully nonperturbative pictures of high-pT energy loss calculations to the first results from LHC. While over-suppressed compared to published ALICE data, parameter-free pQCD predictions based on the WHDG energy loss model constrained to RHIC data simultaneously describe well the preliminary CMS hadron suppression, ATLAS charged hadron v2, and ALICE D meson suppression; we also provide for future reference WHDG predictions for B meson RAA. However, energy loss calculations based on AdS/CFT also qualitatively describe well the RHIC pion and non-photonic electron suppression and LHC charged hadron suppression. We propose the double ratio of charm to bottom quark RAA will qualitatively distinguish between these two energy loss pictures.Comment: 4 pages, 3 figures. Proceedings for Quark Matter 201

    Thermoregulation in rats: Effects of varying duration of hypergravic fields

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    The effects of hypergravitational fields on the thermoregulatory system of the rat are examined. The question underlying the investigation was whether the response of the rat to the one hour cold exposure depends only upon the amplitude of the hypergravic field during the period of cold exposure or whether the response is also dependent on the amplitude and duration of the hypergravic field prior to cold exposure. One hour of cold exposure applied over the last hour of either a 1, 4, 7, 13, 19, 25, or 37 hr period of 3G evoked a decrease in core temperature (T sub c) of about 3 C. However, when rats were subjected concurrently to cold and acceleration following 8 days at 3G, they exhibited a smaller fall in T sub c, suggesting partial recovery of the acceleration induced impairment of temperature regulation. In another series of experiments, the gravitational field profile was changed in amplitude in 3 different ways. Despite the different gravitational field profiles used prior to cold, the magnitude of the fall in T sub c over the 1 hr period of cold exposure was the same in all cases. These results suggest that the thermoregulatory impairment has a rapid onset, is a manifestation of an ongoing effect of hypergravity, and is not dependent upon the prior G profile

    Effect of altered gravity on temperature regulation in mammals: Investigation of gravity effect on temperature regulation in mammals

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    Male, Long-Evans hooded rats were instrumented for monitoring core and hypothalamic temperatures as well as shivering and nonshivering thermogenesis in response to decreased ambient temperature in order to characterize the nature of the neural controller of temperature in rats at 1G and evaluate chronic implantation techniques for the monitoring of appropriate parameters at hypergravic fields. The thermoregulatory responses of cold-exposed rats at 2G were compared to those at 1G. A computer model was developed to simulate the thermoregulatory system in the rat. Observations at 1 and 2G were extended to acceleration fields of 1.5, 3.0 and 4.0G and the computer model was modified for application to altered gravity conditions. Changes in the acceleration field resulted in inadequate heat generation rather than increased heat loss. Acceleration appears to impair the ability of the neurocontroller to appropriately integrate input signals for body temperature maintenance
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